Editing genes has shown a big promise as a way that is not inherited to treat various conditions, including many genetic diseases, even Covid-19. But can the version editing version of the CrisPR gene also help provide long-lasting pain relievers without the risk of addiction associated with prescription opioid drugs? If you want to know about gene edit services you may visit some sites like bosterbio.
In a recent job published in the journal Science Translational Medicine, researchers showed mice that the modified version of the CRISPR system could be used to "turn off" genes in critical neurons to block the pain signal transmission. While more research is needed and its approach is still far from being tested in people, findings indicate that this new Crispr based strategy can form the basis in a completely new manner to manage chronic pain.
This new approach to treating chronic pain occurs in Ana Moreno, the first author of the study when he is a PhD. Students at the Mali Prashant Laboratory supported by NIH, University of California, San Diego. Mali has studied various kinds of novel and cell genes. While reading both, Moreno landed on a paper about mutations in genes that encode the protein that increased the pain in the spinal neuron called NAV1.7.
Moreno reads that children born with mutations in losses in this gene have rare conditions known as the default insurgence of pain (CIP). They really don't feel and respond to pain. Even though these children often fail to recognize serious injuries because there is no pain to remind them, they have no real physical effects of these conditions.